In a recent available Science Daily news explained how new therapy uses synthetic nanofibers to mimic the natural signalling of a protein that is crucial for cartilage formation and maintenance. In a groundbreaking development, researchers have discovered that “dancing molecules” have the potential to heal cartilage damage, offering a promising new treatment for joint injuries and degenerative diseases like osteoarthritis. These molecules, named for their dynamic motion, are designed to move in a way that mimics the natural interactions within the body’s tissues. By actively engaging with cellular structures, these molecules stimulate the regeneration of cartilage by promoting the growth of new cells and the repair of damaged tissue. This innovative approach not only accelerates the healing process but also improves the quality and function of the regenerated cartilage. The success of this technique in preclinical studies suggests that “dancing molecules” could transform the way we treat cartilage injuries, providing a more effective and less invasive alternative to current treatments. As research progresses, this technology holds significant promise for improving the quality of life for millions suffering from joint damage and related conditions.
The article is summarised as “New therapy uses synthetic nanofibers to mimic the natural signaling of a protein that is crucial for cartilage formation and maintenance. Researchers found that intensifying the motion of molecules within the nanofibers led to more components needed for regeneration. After just four hours, the treatment activated the gene expression necessary to generate cartilage. Therapy could be used to treat osteoarthritis, which affects nearly 530 million people worldwide.” The full article can be accessed here.

A concerning news report in Science Daily reported that Covid-19-causing viruses are widely spreading among wildlife.
COVID-19-causing viruses, specifically SARS-CoV-2, are widely spreading among wildlife, raising significant concerns for both animal and human health. Initially, the virus was predominantly associated with human populations; however, mounting evidence indicates that various wildlife species are susceptible to infection. This cross-species transmission can occur directly through contact with infected humans or their environments. The spread of the virus in wildlife reservoirs not only poses a threat to biodiversity but also creates potential long-term challenges for controlling the pandemic. Infected wildlife could serve as a continuous source of viral transmission, complicating efforts to eradicate the virus and increasing the risk of new variants emerging. The situation underscores the need for comprehensive surveillance and preventive measures in wildlife populations to mitigate these risks and protect both ecological and public health.
According to the report, “Six out of 23 common wildlife species showed signs of SARS-CoV-2 infections in an examination of animals in Virginia, as revealed by tracking the virus’s genetic code”. The full article may be found with the title “Virus that causes COVID-19 is widespread in wildlife, scientists find

A piece of recent news by Tina Hesman Saey in Science News has reported, “A mind-bending parasite may one day deliver drugs to the brain”

The concept of using parasites to deliver drugs to the brain is a fascinating and emerging area of research that holds the potential to revolutionize medical treatments for neurological diseases. Traditionally, delivering drugs to the brain has been a significant challenge due to the presence of the blood-brain barrier (BBB), a selective and protective barrier that prevents many substances from entering the brain from the bloodstream. Researchers are now exploring innovative methods to bypass or exploit this barrier, and one such method involves the use of parasites. Parasites, such as Toxoplasma gondii or certain protozoa, have evolved sophisticated mechanisms to cross the BBB and establish infection in the central nervous system. Scientists are investigating how these natural mechanisms can be harnessed and repurposed for therapeutic drug delivery. This innovative approach could lead to targeted and effective treatments for a range of neurological conditions, overcoming one of the most formidable obstacles in the field of medicine. The article may be accessed with the title “Getting drugs into the brain is hard. Maybe a parasite can do the job”.

As per Technology Networks report “European Medicines Agency (EMA) committee has recommended the refusal of EU marketing authorization for Alzheimer’s drug lecanemab (Leqembi), which was only last year given the green light for approval by the US Food and Drug Administration (FDA)”.

Leqembi (commercial name for lecanemab) is an investigational monoclonal antibody developed for treating Alzheimer’s disease. It targets protofibrils of amyloid-beta, a protein that accumulates abnormally in the brains of individuals with Alzheimer’s, forming plaques that are believed to contribute to neurodegeneration. Leqembi works by binding to these protofibrils and facilitating their clearance from the brain, potentially slowing the disease’s progression. Clinical trials have been conducted to evaluate the efficacy and safety of Leqembi in patients with early Alzheimer’s disease, with some promising results showing a reduction in amyloid plaques and a slower cognitive decline compared to placebo. However, as with many experimental treatments, the safety profile and long-term effects of Leqembi are subjects of ongoing research and debate within the medical community. The therapeutic landscape for Alzheimer’s disease remains one of high interest, given the significant unmet need for effective treatments, and Leqembi represents a beacon of hope for advancing the care and management of this debilitating condition.

The news concluded that “The EMA’s decision will come as a disappointment to many, but there are reasons to remain hopeful. Lecanemab has shown that it is possible to slow down disease progression, and research does work,” said Prof. Tara Spires-Jones, president of the British Neuroscience Association and director of the Centre for Brain Science Discovery at the University of Edinburgh. “Each discovery brings us closer to new and better treatments.” The full article can be seen with title “New Alzheimer’s Drug Rejected by European Medicines Agency Committee”.

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The recent news published in Drug Discovery News with entitled “Circular RNA: a new era in therapeutics”. The article has reported that “Originally regarded as a genetic anomaly or an experimental artifact, circular RNA (circRNA) garnered attention after scientists discovered it had a functional role in human cells. In 2011, Erik Digman Wiklund, then a molecular biology graduate student at Aarhus University and now Circio’s Chief Executive Officer, published the first study describing a functional human circRNA”.  The news quoted Erik that “I forgot about circRNA after that,”. Further, he said, recalling his transition from academia to a business career. A decade later, as circRNA emerged as a promising therapeutic avenue, Wiklund revisited his earlier work, spurred by its newfound popularity. After persuading a former laboratory colleague to join him, they founded a company aimed at developing innovative circRNA medicines for rare diseases, vaccines, and cancer. While circRNA therapeutics are still in early stages, Wiklund’s team has demonstrated circRNA’s superior and enduring protein expression compared to traditional mRNA vectors, hinting at its potential to redefine DNA and virus-based therapies in the future. More about the topic can be accessed here.